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Vol.4 , No. 2, Publication Date: Dec. 21, 2019, Page: 11-29
&description=The research evaluated the association between plastics and endocrine disruptors, as well as the viable and active ways to mitigate the challenges posed by plastic pollution. Consumption of seafood represents one major pathway for the exposure of human to microplastic. Microplastics may pass up to higher levels in food chain. Three likely toxic effects of plastic particles have been put forward: the release of persistent organic pollutants (POPs) adsorbed to the plastics, leaching of plastic additives, and plastic particles themselves. Chemical additives such as bisphenol A (BPA), phthalates, polybrominated diphenyl ethers (PBDE), tetrabromobisphenol A (TBBPA), bisphenol S (BPS), etc., used in plastic production pose several health risks to both humans and animals. Due to the use of some chemical additives during production of plastics, plastics have potentially risk and harmful effects that could be carcinogenic or encourage endocrine disruption. Some of the chemical additives are used as phthalate plasticizers (phthalates) and brominated flame retardants. By biomonitoring, chemicals in plastics, such as phthalates and BPA, have been identified in human population. Humans are exposed to the chemicals through the skin, nose, or mouth. Bisphenol A (BPA) can disrupt normal, regular physiological levels of sex hormones. Recent studies suggest that BPS also has endocrine disrupting properties, just like BPA. The presence of a hydroxy group on the benzene ring makes bisphenol S and bisphenol A endocrine disruptors. A widespread concern about phthalate exposure is the possibility that it is the cause of a drop in male fertility. Some recent studies suggest that tetrabromobisphenol A may be an endocrine disruptor and immunotoxicant. As an endocrine disruptor, tetrabromobisphenol A may interfere with both estrogens and androgens. There is also growing concern that PBDEs share the environmental long life and bioaccumulation properties of polychlorinated dibenzodioxins. It is not known if PBDEs can cause cancer in people, although liver tumors developed in rats and mice that ate extremely large amounts of decaBDE throughout their lifetime. The use of biodegradable plastics, policymaking, institutional arrangements, plastic waste collection, promotion of non-usage and lessening usage, incineration, use of the enzyme PETase, and creating awareness, in addition to banning have been the active ways for the management of plastic pollution.&picture=http://www.aascit.org/aascit/decorators/img/journal/809.jpg)
| [1] | Awuchi Chinaza Godswill, Department of Physical Sciences, Kampala International University, Kampala, Uganda. |
| [2] | Awuchi Chibueze Godspel, Department of Environmental Management, Federal University of Technology Owerri, Owerri West, Nigeria. |
The research evaluated the association between plastics and endocrine disruptors, as well as the viable and active ways to mitigate the challenges posed by plastic pollution. Consumption of seafood represents one major pathway for the exposure of human to microplastic. Microplastics may pass up to higher levels in food chain. Three likely toxic effects of plastic particles have been put forward: the release of persistent organic pollutants (POPs) adsorbed to the plastics, leaching of plastic additives, and plastic particles themselves. Chemical additives such as bisphenol A (BPA), phthalates, polybrominated diphenyl ethers (PBDE), tetrabromobisphenol A (TBBPA), bisphenol S (BPS), etc., used in plastic production pose several health risks to both humans and animals. Due to the use of some chemical additives during production of plastics, plastics have potentially risk and harmful effects that could be carcinogenic or encourage endocrine disruption. Some of the chemical additives are used as phthalate plasticizers (phthalates) and brominated flame retardants. By biomonitoring, chemicals in plastics, such as phthalates and BPA, have been identified in human population. Humans are exposed to the chemicals through the skin, nose, or mouth. Bisphenol A (BPA) can disrupt normal, regular physiological levels of sex hormones. Recent studies suggest that BPS also has endocrine disrupting properties, just like BPA. The presence of a hydroxy group on the benzene ring makes bisphenol S and bisphenol A endocrine disruptors. A widespread concern about phthalate exposure is the possibility that it is the cause of a drop in male fertility. Some recent studies suggest that tetrabromobisphenol A may be an endocrine disruptor and immunotoxicant. As an endocrine disruptor, tetrabromobisphenol A may interfere with both estrogens and androgens. There is also growing concern that PBDEs share the environmental long life and bioaccumulation properties of polychlorinated dibenzodioxins. It is not known if PBDEs can cause cancer in people, although liver tumors developed in rats and mice that ate extremely large amounts of decaBDE throughout their lifetime. The use of biodegradable plastics, policymaking, institutional arrangements, plastic waste collection, promotion of non-usage and lessening usage, incineration, use of the enzyme PETase, and creating awareness, in addition to banning have been the active ways for the management of plastic pollution.
Keywords
Plastic Wastes, Endocrine Disruptors, Bisphenol A, Phthalates, Bisphenol S, PBDEs, TBBPA, Microplastics
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